Check your knowledge!
In this section you will find every month three sets of statements concerning tuberculosis and general pediatrics
One statement is true and you are welcome to indicate it to us!

To see what other visitors answered so far after selecting the false statements, press the button "Submit"!

With every new set of questions we will also make available the correct answers and some comments on them.
We hope you enjoy this training and self-review process!

Question 22
Check the false statement.

1. Lymphohematogenous spread takes place before delayed hypersensitivity develops.
2. Mild hypersensitivity reactions in otherwise healthy children are a sign of self-limited infection, whereas intense reactions (larger skin reactions after Mantoux administration) are observed when infection is not controlled and disease develops.
3. Reactivation in the lungs during adolescence and adulthood takes place in the Simon foci and NOT in the primary lesion.
4. Simon foci develop usually in the lower lobes.
5. Miliary tuberculosis is a result of lymphohematogenous dissemination in children and reactivation of old lesions in adults.

Question 23
Check the false statement.

1. The primary lesion(s) may be visible (if calcified) on xray 6-9 months after infection.
2. Most of the radiological findings in tuberculosis are defined by describing the position and histological alterations of affected lymph nodes.
3. Infants and preschoolers have a higher probability than older children of developing secondary pulmonary complications (atelectasis etc).
4. Bronchial restriction from enlarged lymph nodes usually leads to segmental or lobar collapse.
5. Endobronchial lymph node rupture and the resulting pneumonia usually do not cause bronchiectasis. A persistent pneumonia by common bacteria shows a higher predilection for bronchiectasis.

Question 24
Check the false statement.

1. The peak incidence of tuberculous meningitis is in children 6-10 yrs old.
2. Tuberculous mengitis is often combined with miliary tuberculosis.
3. Tuberculous meningitis evades diagnosis during the first stage (I), and this leads to increased morbidity and mortality. This is true for both developed and developing countries.
4. Tuberculous meningitis may develop 3-5 months after infection.
5. Rupture of a parenchymal (or less often meningeal) lesion in the subarachnoid space leads to tuberculous meningitis.

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